Address: (Linghao Road) No.2 Street, No.20 Economic Development Zone, Hangzhou
PACLITAXEL ELUTING CORONARY STENT With RX delivery system Innovative bifurcation stent system: One profiled balloon with diameters corresponding to dimensions of the coronary bifurcation vessels Unique configuration of the delivery system ensuring safety and efficacy during the stent......Send Inquiry
PACLITAXEL ELUTING CORONARY STENT
With RX delivery system
Innovative bifurcation stent system:
One profiled balloon with diameters corresponding to dimensions of the coronary bifurcation vessels
Unique configuration of the delivery system ensuring safety and efficacy during the stent implantation procedure
Delivery system minimizes the negative effects of procedure, protecting the carina tip from being crushed or damaged
Large cell of the stent in place of side branch entrance gives possibility to enter the SB with any standard size conventional stent
Precision of implantation procedure thanks to three radiopaque markers
ADVANTAGE OF OUR INNOVATED STENT SYSTEM
Non-implanted Nitinol drug-eluting stent system
Drug release rate > 90%; drug transferred within 60 seconds, release time reaches 90 days
Effectively lower the occurrence rate of arterial dissection (currently 20% or so)
-Diagram of vessel wall injury caused by common balloon torsion
-Diagram of vessel wall injury caused by radial stress of common
-Diagram of vessel wall injury caused by longitudinal stress of common balloon
-Stress distribution diagram of "Common balloon vs new product"
Our paclitaxel drug eluting stent system takes the ideal definition of Safety - Efficiency - Deliverability for Percutaneous Coronary Intervention (PCI) to a higher level, convinces with its trusted technology and largest size portfolio available provides high end performance with less metallic coverage but reliable outcomes, and offers an optimal solution for improving luminal diameter and reducing in-stent restenosis when treating coronary artery lesions.
How does paclitaxel work?
Paclitaxel works by stabilizing microtubules, cell elements that are responsible for cell division, rendering them nonfunctional. The drug has unique multifunctional effects that inhibit the restenotic process. It promotes the polymerization of stable nonfunctioning mictrotubules and thus inhibits all microtubule-dependent activities. As a result, it can inhibit multiple cellular components of the restenotic process. In addition, paclitaxel is highly lipophilic, which contributes to more efficient drug transfer off the stent and into the tissue. Lastly, paclitaxel has a dose-dependent inhibitory effect on smooth muscle cell proliferation. Because smooth muscle cells are more sensitive to paclitaxel than endothelial cells, it reduces restenosis without compromising the healing of the arterial wall.